CD69 and cancer: • Activated by CD1d-dependent lipid antigen presentation (49)• CD1d and IL-7 dependent cell maintenance (49, 50)• Higher expression of Bcl-2 and Socs2 (12)• Higher proliferation status (12)• Related to hepatocyte cell death and liver damage (14, 49)• LFA-1, CXCR6, and CD69 for residence and maintenance (41, 51–54)• NKT17: promote cancer cell extravasation (55)