To demonstrate that exon skipping cross-correction via snRNA is effective also with other types of myogenic cells, we co-cultured DMD-U7 TIM and WT TIM cells with two different DMD myogenic cell types, either mesoangioblasts (DMDhMABs) or MyoD-converted Fibroblasts (DMDMyoDERTFbs) derived from DMD patients with the same mutation. The gene discussed is MYOD1; the disease is Duchenne muscular dystrophy.