NFKB1 and diabetic cardiomyopathy: Investigators found that knockdown of Salusin-β effectively inhibited oxidative stress, NF-κB activation, and upregulation of inflammatory responses via NADPH oxidase 2 (NOX2)/ROS/NF-κB signaling in a rat model of diabetic cardiomyopathy, whereas the NF-κB inhibitor Bay 11-7082 attenuated inflammatory responses only.441