To confirm whether IL17 in human cervical cancer cells plays the same role for macrophages as it does in vitro cell lines, we quantified M1 and M2 macrophage populations in normal tissues, primary tumor tissues, and metastatic tissues of cervical cancer with high IL-17A expression using flow cytometry and showed significantly increased CD206 cells and decreased CD86 cells in cervical cancer cells (Fig. 3H, I). This evidence concerns the gene CD86 and neoplasm.