In addition to those specifically regulated in GBM, URs activated by DHA and shared between GBM and MM-BM cell lines and/or between GBM and MM cell lines revealed a large set of immune-related activated URs (e.g., cytokines, IFNs, JAK-STAT, Toll-like receptor, and TNF-α superfamily) (see Fig. 6, panels #1 A, B; Additional file 7: Tables S6, S7). This evidence concerns the gene SOAT1 and Miyoshi myopathy.