Our levels of NKG2C+ NK cells are much lower than what one would expect for an NKG2C+-inducing infection, such as by CMV; thus, they likely represent baseline levels, such as baseline inflammation, rather than the capacity to respond to, for example, Epstein-Barr virus (EBV), whose control by NKG2C+ NK cells has been linked to reduced prevalence of multiple sclerosis.82 Here, KLRC2 is linked to multiple sclerosis.