Interestingly, we observed a far greater incidence of co-occurring NAT2 UAs and ATP7B 832 R/R homozygosity in AT-DILI patients (13.9%, 10/72) than in treatment-tolerant controls (0.5%, 1/205; P = 0.017; Fig. 2b). This evidence concerns the gene ATP7B and ataxia telangiectasia.