SLITRK1 and Alzheimer disease: Further, Ptprd, Akap12, Cadm1, Myo5a, Slitrk1, and Pdpn were involved in “synaptic” related biological processes, with four of them (Ptprd, Akap12, Cadm1, and Myo5a) related to the Schaffer collateral‐CA1 synapse, one of the areas with early dysfunction in AD.41