‘s research demonstrated the SUVmax of lesions with PD-L1 expression degrees ≥ 50% was significantly higher than that of lesions with PD-L1 expression degrees < 50%, and the SUVmax was significantly higher in the tumor with KRAS mutation than in those without KRAS mutations (4); 18F-FDG uptake was closely correlated with the expression of glucose transporters in varying amounts in PSC (13); Patients with high 18F-FDG uptake have the significantly higher mean scoring of Glut1, Glut3, VEGF positivity and number of microvessels (CD34) than those with low 18F-FDG uptake (8). The gene discussed is KRAS; the disease is neoplasm.