AXIN2 and colorectal carcinoma: Although CK1ε may mediate the stability of AXIN1 and AXIN2 via a similar mechanism, our real‐time PCR results demonstrated that silencing CK1ε or treatment with CK1δ/ε inhibitors downregulated the mRNA expression of AXIN2 through inhibiting Wnt/β‐catenin signaling in CRC cells.