Several members of the DGK family have been implicated in CRC.34 In particular, DGKG plays a role in DNA methylation in CRC tumors, suggesting it is an early event during CRC tumorigenesis.34 In line with altered PC metabolism in CRC,35 we also identified overexpression of LPCAT2, which is part of the Lands cycle and is involved in the reacylation of LPC into PC. This evidence concerns the gene LPCAT2 and colorectal carcinoma.