Therapeutic effect on mouse psoriatic inflammation by downregulating oxidative and inflammatory mediators in neutrophils and dendritic cells has been reported by Al‐Harbi et al.2T helper 17 (Th17) cells and its effective cytokine interleukin (IL)‐17A play a pivotal pathogenic role in the development of psoriasis, and IL‐17A inhibitors also present excellent therapeutic effects for moderate‐to‐severe plaque psoriasis.3 This evidence concerns the gene IL17A and psoriasis.