These differentially expressed bacterial sRNAs were predicted to interact with several human 3’UTRs in bronchiolitis and potentially lead to the activation of the IL-8 and IL-6 pathways and inhibition of the IL-17A in RSV-only cases, and thus the inhibition of the IL-8 and IL-6 pathways and activation of the IL-17A pathway in RV-bronchiolitis. The gene discussed is IL6; the disease is bronchiolitis.