As expected, we also observed lower expression of cytotoxic, cell cycle and energy metabolism signatures, upregulation of genes related to mitochondrial function, NK receptor (KLRC2, KLRC3) and FYN, inhibited cell cycle and metabolism pattern and activated mitochondrial dysfunction in Te cells from the MG patients at baseline, compared with Te cells from lymphoma patients in external cohorts (Figs. 6C and EV4B). This evidence concerns the gene KLRC2 and myasthenia gravis.