This could be corroborated by our proteomics functional analysis, where we found, for instance, a higher abundance of HADH, PLOD3 and ACAT1 (lysine degradation) and PFKL, NTRK1, PDHB and PDHA1 (central carbon metabolism in cancer) in control piglets compared to UL. The gene discussed is PLOD3; the disease is cancer.