However, genome‐wide association studies have shown the genetic variants that may contribute to the risk of IPF, such as telomerase reverse transcriptase (TERT), regulator of telomere elongation helicase 1 (RTEL1), KLF transcription factor 15 (KLF15), mitotic arrest deficient 1 like 1 (MAD1L1), SFTPC, and mucin 5B (MUC5B).157, 158, 159, 160. The gene discussed is MAD1L1; the disease is idiopathic pulmonary fibrosis.