We correlated the expression of DEGs from our time course analysis of decidualization with the top 50 genes indicating the key branching points between a decidual and non-decidual cells from the study by Lucas ES et al.76 With this correlation we found that cells from endometriosis donors displayed higher expression signatures associated with immune surveillance (TIMP3, CXCL14), cellular stress (CRYAB, GLRX, HSD11B1), increased iron storage (FTL, SCARA5), and partial alignment with genes characteristic of decidual senescence (ADAMTS5, ABI3BP, CEMPI and COL14A1). The gene discussed is COL14A1; the disease is endometriosis.