TBK1 and neoplasm: In contrast to the SCCVII and Pan02 cells, JAWSII cells displayed a fully activated STING pathway even when only the lowest DMXAA dose was used, as indicated by the significant levels of P‐TBK1 and P‐IRF3 (JAWSII, Fig. 1A), suggesting that the activation of the STING pathway in both tumor cells occurred only to a much lesser extent than in non‐malignant cells.