In this sense, B1R expression is stimulated by IL-1β, TNF, and IFN-γ [40,41,42] and may be fostered in inflammatory and pathological circumstances, such as tissue or vascular injuries, ischemia, and diabetes, as well as upon infection by certain pathogens, including viruses [14,15,43,44,45]. This evidence concerns the gene BDKRB1 and infection.