In other models, infection of HUVECs with P. gingivalis W83 [53] and ATCC 33277 [54] reduced the soluble levels of eNOS mediated by the induction of the activation of PPARγ in ECs [53] and the action of bacterial HSP 60 [55]; however, in the model in this work, these data cannot be adequately related as the soluble concentration of eNOS was not measured. This evidence concerns the gene NOS3 and infection.