This induces several mechanisms, such as G2/M arrest, apoptosis, the activation of caspases 3, 8, and 9, and DNA damage occurrence (γH2AX and 8-OHdG) to a greater extent in oral cancer cells than in normal cells (Figure 2, Figure 4, Figure 5, Figure 10 and Figure 11). This evidence concerns the gene CASP3 and lip and oral cavity carcinoma.