Afterwards, we set up a CRISPR/cas9 gene editing system that focuses on the LMP1 fragment in LMP1 (latent membrane protein 1, related to Epstein–Barr virus (EBV) infection)-overexpressed SCC7 cells (SCC7) so as to simulate an EBV-positive subcloned cell line (C666-1) of human nasopharyngeal carcinoma cell lines [29]; we also conducted pharmacodynamic experiments on mice. This evidence concerns the gene PDLIM7 and nasopharyngeal carcinoma.