Interestingly, many of these cytokines, such as MCP-1, IL-1β, and TNF-α, do overlap with the inflammatory mediators, which are upregulated by lipopolysaccharide (LPS)-induced AKI, demonstrating parallels between SARS-CoV-2-induced AKI and sepsis-induced AKI [99,100]. This evidence concerns the gene CCL2 and acute kidney injury.