LGALS8 and neoplasm: Regarding tumor differentiation grades, the distribution of the Gal-8 immunoexpression was registered in G1 and G3 (each in 36.95%) and G2, in only 26.08%, in our study group, supporting its constant involvement in ovarian carcinogenesis, regardless of the tumor degree of differentiation, either directly or by the possible activation of different proliferative molecular pathways already identified in other tumors [10,11].