Delving into the molecular mechanisms through which α-MSH and MCRs suppress apoptosis in neuronal and associated cell types, in the immortalized hypothalamic tumor cell line, GT1-1, the synthetic analog of α-MSH, NDP-MSH, at a dose of 10−6 M, was shown to inhibit serum deprivation-induced caspase 3 activation. Here, STAMBP is linked to neoplasm.