Indeed, FOXP1 overexpression decreases NF-κB p65 protein expression, while EBV-miR-BART11 overexpression or FOXP1 knockdown increases NF-κB p65 expression, indicating the role of NF-κB in the crosstalk between NPC cell and TAMs in response to inflammation and tumor development [181]. This evidence concerns the gene FOXP1 and nasopharyngeal carcinoma.