It can also ameliorate the symptoms of atopic dermatitis by inhibiting the production of pro-inflammatory cytokines, e.g., TNF-α (tumour necrosis factor-alpha), and IFN-γ (interferon gamma) [36] and inhibit the development of acute gastritis by affecting the levels of pro-inflammatory response elements—by decreasing the inducible nitric oxide synthase (iNOS), cyclooxygenase-2 (COX-2), TNF-α, IL-1β and IL-6 [37]. This evidence concerns the gene IFNG and atopic eczema.