NFKB1 and tuberculosis: Since TLR stimulation after Mtb infection can activate the NF-κB and MAPK pathways and participate the anti-tuberculosis immune response [41], while Mtb can inhibit these two signaling pathways and promote Mtb survival through bacterial components such as PPE36 to suppress host innate immunity [42], we analyzed the effects of these signaling pathways on CEBPB phosphorylation.