NLRP3 and diffuse large B-cell lymphoma: For DLBCL, which is the most studied lymphoma subtype regarding the NLRP3 inflammasome, it can be proposed that NLRP3 inflammasome activation is driven by two main mechanisms: (i) lymphoma cell-intrinsic features favoring an autonomous signal and (ii) bidirectional interactions between lymphoma cells and the TME.