UCA1 and neoplasm: GA-MSCs enhance tumor growth and invasion through different mechanisms: (1) CXCL14, expressed by GA-MSCs, is positively correlated with upregulation of long non-coding RNA UCA1 and downregulation of miR-182 in GB cells, which drives glycolysis and tumor cell invasion through the UCA1/miR-182/PFKFB2 axis [46].