Lastly, STAT6 is activated in response to IL-4 and IL-13 [23,24] via phosphorylation at Tyr641 [25] and, in Daudi cells (also called Burkitt lymphoma) after stimulation with IFN-α, STAT6 forms an unusual heterodimer with STAT5 protein [26]. This evidence concerns the gene STAT6 and Burkitt lymphoma.