Furthermore, the rs7173743 intergenic variant, located 40 kb upstream of the transcription start site of the ADAMTS7, seems as well to be a promising candidate risk factor, as previous GWAS studies suggested an association of this polymorphism with CAD [13] and showed its significant role as a risk factor of unstable atherosclerotic plaque occurrence [14]. This evidence concerns the gene ADAMTS7 and coronary artery disorder.