CD8A and primary cutaneous T-cell non-Hodgkin lymphoma: Transcriptomic studies showed a distinctive pathophysiology involving CD8 T-cells and CXCL9 and CXCL11 expression, two macrophage-derived chemokines that were upregulated in the skin of MARs, and the key players were macrophage–lymphocyte mediators in the context of CTCL.