Furthermore, persistent inflammation after invasion, resulting from the production of proinflammatory cytokines such as interleukin-6 (IL-6), tumor necrosis factor alpha (TNF-α), interferon-gamma (IFN-γ), and CXC chemokine ligand 10 (CXCL10), appears to contribute to the development of post-COVID-19 OD, as it can directly damage the OE or interfere with cell signaling processes [13,33]. This evidence concerns the gene CXCL10 and COVID-19.