The hypothesis that this could be the cause of hypo/anosmia derived from COVID-19 has been questioned by numerous studies that have highlighted the absence of angiotensin-converting enzyme 2 (ACE2) and transmembrane serine protease 2 (TMPRSS2), the key factors for the virus’s entry into the cell [23,24]. This evidence concerns the gene TMPRSS2 and COVID-19.