The authors explained that the Toxoplasma latent infection leads to the release of Interferon- γ (IFN) which induces the production of an enzyme key to Toxoplasma replication, releasing kynurenic acid as a metabolic product of said replication, which is an antagonist molecule for the glutamate N-methyl-D-aspartate (NMDA) receptor, which plays a key role in schizophrenia neuromodulation [36]. The gene discussed is IFNA1; the disease is schizophrenia.