Helminth infections are widely known to lead to T helper type 2 (Th2) responses typified by high levels of the profile of cytokines they induce, such as IL-4, IL-5, IL-10, and IL-13, accompanied by high levels of characteristic antibodies such as IgG1 and IgE, as well as increased numbers of eosinophils, goblet cells, and mast cells [1]. This evidence concerns the gene IL4 and helminthiasis.