This finding aligns with previous studies indicating that commonly up-regulated pathways in the pathogenesis of IBD (both CD and UC) include the toll-like receptor pathway, NOD-like receptor signaling pathway, cytokine–cytokine interaction, chemokine signaling, intestinal immune network for IgA production, complement and coagulation cascade, and cell adhesion molecules [24,26,47]. Here, CD79A is linked to inflammatory bowel disease.