Histologically, tumor cells had a similar appearance, and by gene expression, these tumors showed activation of some of the same pathways, including EMT, lysosome, cell proliferation (E2F, Myc, cell cycle, DNA replication, mitotic spindle), and inflammation (inflammatory response, TNF-α/NF-κB, allograft rejection). This evidence concerns the gene NFKB1 and neoplasm.