Along with the abundant evidence of endothelial progenitor cells [30] and sFlt-1 (soluble fms-like tyrosine kinase 1) and PlGF (placental growth factor) [31] in the placental dysfunction, our finding suggested potentially synergetic pathology of lipid alternation in postpartum hemorrhage and placental abruption among preeclamptic women. This evidence concerns the gene PGF and placental abruption.