PRX and cancer: [31] To evade senescence, apoptosis, necrosis, and ferroptosis, cancer cells strategically modulate multiple antioxidant defense mechanisms for the proper elimination of increased levels of ROS/RNS to establish new redox homeostasis.[32] For example, increased expression or activity of antioxidative‐related enzymes such as SOD and its mimics, CATs, GPXs, and Prx have been demonstrated in many different cancers.