MYO7A and nodular sclerosis classical Hodgkin lymphoma: However, for other variants their zygosity status might support their involvement in NSHL pathogenesis including c.3641G > A and c.6503T > G in MYO15A [reported by the Department of Genetics, SQUH-Genetics Sultan Qaboos University Hospital in Oman to cause NSHL in homozygous state (53)], c.599C > T in WHRN, c.2476G > A and c.4696A > T in MYO7A (compound heterozygous), c.− 182G > A and c.617-3_617-2dup in TMPRSS3 (compound heterozygous), and c.98G > A in OTOF. Moreover, two of those variants, c.599C > T in WHRN and c.98G > A in OTOF indicated a damaging impact on the protein structure.