Did they like the similar studies that have been performed on EAE, diabetes, and systemic lupus erythematous disease [32]; (6) Which subsets of autoantigen-specific CD8+ Tregs play a regulatory role in MG/EAMG among CD8+CD28, CD8+CD122+ [106], CD8+ CD122+ CD49d+ cells [88–91], or CD8+ CD122hi Ly49+ cells [92–105], which have been well studied in other diseases. Here, KLRA1P is linked to myasthenia gravis.