RAD54B and gastric cancer: The results showed that the number of brunching points of HUVECs was significantly diminished after HUVECs were cultured with supernatants form cultured AGS and MKN45 cells with the knockdown of RAD54B, while that was prominently increased after HUVECs were cultured with supernatants form cultured AGS and MKN45 cells with the overexpression of RAD54B (Figure 4), indicating that RAD54B promoted the tube formation of gastric cancer cells.