All populations up-regulated IL1B, IL6, IL10, IL18, IL22, CXCL8, CCL4, TLR1, ACOD1, TLR4, and TLR7 when infected with evolved MG (VA2013), which would propagate inflammation and facilitate pathogen transmission through pathological mycoplasmal conjunctivitis (15, 36). This evidence concerns the gene CCL4 and myasthenia gravis.