CCL5 and neoplasm: Cafestol (30 μM) and kahweol (30 μM) acetate inhibited proliferation and migration by inducing apoptosis, inhibiting epithelial–mesenchymal transition, and decreasing androgen receptor, resulting in a decrease in nuclear androgen receptor in androgen receptor‐positive cells. Furthermore, kahweol acetate and cafestol inhibited CCR2 and CCR5 while increasing their ligands, CCL2 and CCL5. The xenograft study found that taking kahweol acetate and cafestol orally significantly reduced tumor growth