HuR is known to promote carcinogenesis and treatment resistance in the different cancers, such as breast cancer, colon cancer and glioma, by interacting with a subset of mRNAs related to hormone receptors, inflammation and proliferation [14] because HuR post transcriptionally regulates proliferation, apoptosis, senescence, inflammation and immunity by binding and stabilizing the target gene mRNA [15]. This evidence concerns the gene ELAVL1 and colonic neoplasm.