AKT1 and cancer: In cancer cells, the quiescence program of dormancy is induced by p38/MAPK activation,81 extracellular signal‐regulated kinase (ERK)101 and phosphoinositide 3‐kinase (PI3K)/protein kinase B (AKT) inactivation,102 as well as by decreasing glycolysis103 and glucose uptake.104, 105