Figure 4(a)–4(c) (Supplementary 2) showed that QFTL significantly downregulated the protein expression levels of α-SMA and MMP12 in BLM-induced pulmonary fibrosis rats (P < 0.05). Similarly, the immunofluorescence assay revealed that QFTL could reduce the BLM-induced expression level of α-SMA in rats (Figures 4(d) and 4(e)). When comparing the expression levels of α-SMA protein between the CQ + QFTL group and the QFTL group, we noticed a visible difference in the values between the two groups, and further statistical results showed significant differences (P < 0.05). This evidence concerns the gene ACTA1 and pulmonary fibrosis.