Furthermore, Sacreas et al [19] suggested that the fibrotic process of RAS mirrors that of idiopathic pulmonary fibrosis and may be driven by mesothelial-to-mesenchymal transition, characterized by differentiation of pleural mesothelial cells into myofibroblasts after stimulation by TGFβ. Here, TGFB1 is linked to idiopathic pulmonary fibrosis.