Then, the mRNA stability of SLC7A11 was examined in HCC cells treated with 50 mM actinomycin D. SLC7A11 transcription was inhibited after LINC00942 silencing compared with that in the β-actin group (Fig. 5E; p = 0.0030 in HepG2 cells, p = 0.0012 in HuH-7 cells, n = 3). This evidence concerns the gene SLC7A11 and hepatocellular carcinoma.